Link between Fructose & Obesity

It is sugar contained in large quantities, in fruits and honey, and could induce obesity. It would send a message to the brain to trigger hunger.

Compared to glucose, which gives a message of satiety, fructose is worrying, because the obesity rate retained in North America would result from the high rate of fructose contained in food. According to a study, it could be that fructose has little effect on the regions of the brain concerned with the regulation of appetite. In industrial food we find fructose, because with its more sweetening power, it transforms the texture of products.

In sodas and colas, corn syrup and sucrose fructose is ubiquitous, as well as many foods such as pastries and sauces, not to name them all.

Fructose is assimilated and transformed differently than glucose, despite the fact that it is a monosaccharide. The assimilation of fructose, stimulates the production of insulin in the body less than that of glucose, because this hormone activates satiety and reduces the need for food. According to some experiments on laboratory mice, the ingestion of fructose creates in the brain, a feeling of hunger, a fortiori, glucose revealed the opposite

According to some studies, such as that of JAMA, the consumption of fructose reduced the blood flow of the ippocampus, inhibiting the feeling of hunger. Fructose encourages increased food intake. Excessive consumption of calories promotes overweight, and therefore that of fructose. It is necessary to privilege glucose to fructose, if one wishes to be able to control his weight. The correlation between the ingestion of glucose and the resulting satiety has been demonstrated, which confirms that fructose is indeed a vector of hunger.

The consumption of glucose, decreases the activation of the hypothalamus, the insula and the striatum of the brain. In this case, the ingestion of glucose activates the connections of the hypothalamo-striatal network, thus the feeling of satiety. The ingestion of fructose considerably reduces the doses of insulins which are satiety information hormones. From the moment we absorb glucose, we are quickly satiated, but with fructose hunger returns.

The link of cause and effect is clearly established, fructose causes feelings of hunger and persuades that it is necessary to consume food again, even if physiologically this is an absurdity.